Shock Hemodynamics

Each type of shock has a distinct hemodynamic fingerprint. Learn the profiles — and use the chart — to tell them apart at a glance.

🩺 Reviewed by our Editorial Team⏱ 2 min read🗓 Updated July 2026

What is shock?

Shock is a state of inadequate tissue perfusion — oxygen delivery fails to meet demand. Its four broad categories each disturb the pressure–flow–resistance balance differently, and hemodynamic monitoring (CVP, PCWP, cardiac output, and SVR) reveals which one you are dealing with.

The shock hemodynamics chart

This is the classic comparison — memorise the pattern, not just the rows:

TypeCVP / RA (preload)PCWPCardiac outputSVRSvO₂
Hypovolemic
Cardiogenic
Obstructive↓/↑*
Distributive (e.g. septic)↓/normal↓/normal↑/normal**↑ (early)

*Depends on the cause (tamponade vs pulmonary embolism). **"Warm" distributive shock has high output early; it can fall late.

The four profiles explained

Clinical pearls

Quick keys. Low SVR points to distributive shock; high PCWP with low output points to cardiogenic; low everything with high SVR points to hypovolemia; high CVP with clear lungs and low output suggests obstructive (think tamponade or PE).

Summary

Practise hemodynamics

Test shock, pressures, and waveform questions with explanations.

Practise Hemodynamics →

Frequently asked questions

What is the hemodynamic profile of each type of shock?

Hypovolemic: low CVP/PCWP, low output, high SVR. Cardiogenic: high PCWP/CVP, low output, high SVR. Obstructive: high CVP, low output, high SVR. Distributive: low SVR with high or normal output early.

What is a shock hemodynamics chart?

A table comparing CVP, PCWP, cardiac output, SVR, and SvO₂ across the four shock types so you can pattern-match the diagnosis.

How do you tell cardiogenic from hypovolemic shock?

Cardiogenic shock has high filling pressures (PCWP/CVP) with low output; hypovolemic shock has low filling pressures with low output.

Which shock has a low SVR?

Distributive shock (such as septic, anaphylactic, or neurogenic) causes vasodilation and a low systemic vascular resistance.

Sources & further reading

External links are provided for reference; always confirm current details with the official source.

RCIS Practice Test Editorial Team

Our content is written and reviewed by contributors with cardiovascular and allied-health backgrounds, grounded in standard references and the official CCI exam domains. Educational use only — not medical advice. See our editorial policy.