Constrictive vs Restrictive vs Tamponade

Three conditions raise the heart's filling pressures and look almost identical at the bedside — constrictive pericarditis, restrictive cardiomyopathy, and cardiac tamponade. The waveforms and a few respiratory tricks are how the cath lab tells them apart.

🩺 Reviewed by our Editorial Team⏱ 5 min read🗓 Updated July 2026

The short answer

Constriction = a stiff pericardium: prominent y descent, dip-and-plateau, equalised pressures, Kussmaul's sign, and — the clincher — discordant RV/LV systolic pressures with breathing. Restriction = a stiff myocardium: same dip-and-plateau, but LVEDP exceeds RVEDP by >5 mmHg and the ventricles move concordantly. Tamponade = fluid under pressure: blunted y descent, equalised pressures, and marked pulsus paradoxus.

FeatureConstrictionRestrictionTamponade
RA y descentProminent (steep)ProminentBlunted / absent
Dip-and-plateauYesYesNo
LVEDP vs RVEDPEqual (≤5 mmHg)LV > RV (>5 mmHg)Equal
Respiratory RV/LV systoleDiscordantConcordantConcordant
Kussmaul's signPresentVariableAbsent
Pulsus paradoxus±NoMarked
PA systolicUsually < 55 mmHgOften > 55 mmHgNormal / low

See the three side by side

Open the interactive atlas below and switch between Constrictive pericarditis, Restrictive cardiomyopathy, and Cardiac tamponade in the condition list. Turn on Breathing and Connect peaks for constriction to watch the RV and LV systolic peaks move in opposite directions — the discordance that clinches the diagnosis.

Open the atlas full-screen →

What each condition is

The three differ by where the problem sits:

All three raise filling pressures, which is why they mimic one another — and why the fine detail of the waveforms matters so much.

Constriction vs restriction: the key differences

These two share the most overlap — both show a prominent y descent and a dip-and-plateau. Three findings separate them:

  1. Filling-pressure equalisation. In constriction the diastolic pressures of all four chambers land within about 5 mmHg of each other. In restriction the left ventricular end-diastolic pressure runs more than 5 mmHg above the right, because the left ventricle is usually the stiffer of the two.
  2. Ventricular interdependence. This is the most specific sign. Because a rigid pericardium fixes the heart's total volume, constriction makes the ventricles compete — on inspiration the RV systolic pressure rises while the LV systolic pressure falls (discordance). In restriction the shell is intact, so the two ventricles rise and fall together (concordance).
  3. Pulmonary pressures. Restriction tends to drive the PA systolic pressure higher (often above 55 mmHg); constriction usually keeps it lower.
Bottom line. If the ventricular systolic peaks move in opposite directions with breathing, think constriction — a surgically treatable disease. If they move together and the LVEDP outstrips the RVEDP, think restriction.

Constriction vs tamponade: the y descent

Constriction and tamponade both equalise the filling pressures, but the atrial waveform gives them away:

Our dedicated guide on cardiac tamponade hemodynamics goes deeper on the blunted y descent and Beck's triad.

How the cath lab confirms it

The definitive test is a simultaneous left- and right-ventricular pressure recording with a respirometer. The operator watches the two systolic pressures across the breathing cycle:

You can rehearse the whole study — access, right-heart catheterisation, reading the M/W wave, the dip-and-plateau, Kussmaul's sign, and the discordance — in our step-by-step interactive tools, then test yourself on scenario questions.

Test yourself on the look-alikes

Free scenario questions on tamponade, constriction, and restriction.

Practise Scenarios →

Frequently asked questions

What is the main difference between constrictive pericarditis and restrictive cardiomyopathy?

In constriction the diastolic pressures equalise and the ventricles show respiratory discordance (RV rises as LV falls). In restriction the left ventricular end-diastolic pressure exceeds the right by more than 5 mmHg and the ventricles move concordantly. Constriction is a pericardial disease; restriction is a myocardial one.

How do you tell constriction from tamponade?

Both equalise filling pressures, but constriction preserves a prominent, steep y descent and Kussmaul's sign, while tamponade blunts the y descent, lacks Kussmaul's sign, and shows marked pulsus paradoxus.

What is ventricular discordance?

During inspiration in constriction, the right ventricular systolic pressure rises while the left ventricular systolic pressure falls — they move in opposite directions. This 'discordance' (systolic-area index >1.1) is the most specific catheterisation sign of constriction.

What is Kussmaul's sign?

A failure of the jugular venous (right atrial) pressure to fall — or a paradoxical rise — during inspiration. The rigid pericardium in constriction prevents the normal inspiratory drop. It is usually absent in tamponade.

Why is the y descent blunted in tamponade but prominent in constriction?

In tamponade the fluid compresses the heart so it cannot expand in early diastole, flattening the y descent. In constriction the ventricle still fills rapidly early on before hitting the stiff shell, so the y descent is steep and prominent.

Is constrictive pericarditis curable?

Often, yes. Unlike restrictive cardiomyopathy, constriction is frequently treated surgically by removing the thickened pericardium (pericardiectomy), which is why distinguishing the two matters so much.

What pulmonary artery pressure suggests restriction over constriction?

A pulmonary artery systolic pressure above roughly 55 mmHg favours restrictive cardiomyopathy; constriction usually keeps the PA systolic pressure lower.

Do all three conditions show a dip-and-plateau?

No. Constriction and restriction both show the dip-and-plateau (square-root sign) in the ventricular tracing, but tamponade does not.

Sources & further reading

External links are provided for reference; always confirm current details with the official source.

RCIS Practice Test Editorial Team

Our content is written and reviewed by contributors with cardiovascular and allied-health backgrounds, grounded in standard references and the official CCI exam domains. Educational use only — not medical advice. See our editorial policy.